- Etiology:
— Combination of immaturity, enteral feeding, ischemia, infection which leads to mucosal damage which leads to intestinal ischemia which leads to necrosis
— Involves any portion of intestine but distal ileum and proximal colon most common - Imaging AXR:
— Early see diffusely dilated air-filled loops of bowel and ileus or asymmetrically focally dilated loop of bowel
— Later see classic triad of pneumatosis intestinalis, portal venous gas, free air
— Fixed bowel loop can be sign of impending perforation
— Some perforations (15-20%) can be walled off due to surrounding inflammation and not show free air on AXR
— Many cases have no AXR findings as some findings of necrotizing enterocolitis on AXR are transient such as pneumatosis intestinalis and portal venous gas
— Bubbly pneumatosis intestinalis = submucosal gas, curvilinear pneumatosis intestinalis = subserosal gas
— Note: Biliary air is more central in liver, portal venous gas is more peripheral in liver
— Note: Portal venous gas when seen with pneumatosis intestinalis is forewarning of more severe bowel ischemia - Imaging US:
— Complicated ascites and free air are signs of perforation
— Portal venous gas appears as echogenic bubbles in portal veins
— Pneumatosis intestinalis - DDX:
- Complications: Strictures in 25% which are more common in colon than small bowel, especially in descending colon
- Treatment: Surgical intervention when there is free air due to bowel perforation
- Clinical:
— Mainly seen in premature infants with feeding intolerance, abdominal distension, bloody stools
— Up to 10% of cases occur in term infants who are stressed (after cardiac surgery)
— Is most common cause of portal venous gas in a neonate
— Is most common cause of pneumatosis intestinalis in children - Note: Stricture should be suspected in an infant with a persistently distended abdomen after treatment for necrotizing enterocolitis
Imaging US:
- US can help when AXR is unremarkable, AXR has paucity of bowel gas or is gasless, AXR shows dilated fixed loop of bowel which leads to check its perfusion and peristalsis
- US most helpful findings – free air, absent peristalsis, complex ascites, focal fluid collections
- Findings:
— Bowel wall thickening (greater than 2.7 millimeters) or thinning (less than 1 millimeters)
— Pneumatosis intestinalis, portal venous gas, ascites, fluid collection, peristalsis, zebra pattern of prominent valvulae conniventes
— US Doppler of superior mesenteric artery and celiac trunk – increased peak systolic velocity, hyperemia or no flow - Progression of bowel wall findings:
— Start with normal bowel wall
— Which leads to bowel distension and hyperemia with or without echogenic mucosa
— Which leads to bowel wall thickening with or without hyperemia, with or without intramural gas with or without loss of gut signature with or without loss of bowel wall distinctness
— Which leads to bowel wall thinning and bowel distension and bowel contents with debris with or without intramural gas
Approach to necrotizing enterocolitis:
- On AXR and US look for:
— 1) Gas – intraluminal bowel gas – dilated pattern, intramural gas, portal venous gas, free gas and 2) calcification – peritoneal, mural, luminal - On US look for:
— 1) Fluid – free fluid, focal fluid or abscess, intraluminal gastrointestinal fluid and 2) bowel wall – thickness and echogenicity, peristalsis (real time), perfusion (Doppler)
Radiology Cases of Necrotizing Enterocolitis
Radiology Cases of Diffusely Dilated Loops of Bowel in Necrotizing Enterocolitis

Radiology Cases of Pneumatosis Intestinalis in Necrotizing Enterocolitis

Radiology Cases of Portal Venous Gas in Necrotizing Enterocolitis

Radiology Cases of Bowel Perforation in Necrotizing Enterocolitis

Radiology Cases of Bowel Stricture After Necrotizing Enterocolitis





Radiology Cases of Enterocutanous Fistula After Necrotizing Enterocolitis

Surgery Cases of Necrotizing Enterocolitis

Surgery Cases of Bowel Stricture After Necrotizing Enterocolitis
